RT-107
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Evaluation of myocardial lesion
after radiofrequency catheter ablation using in-111-antimyosine antibodies: preliminary
results
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X. Vinolas, I. Carrió*, R. Oter,
O. Aslan, M. Ballester, P. Torner, M.C. Varela, A. Bayés de Luna.
Cardiology and Cardiac Surgery Department,
*Nuclear Medicine Department, Hospital de Sant Pau, Barcelona, Spain
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References
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1.
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Nath S, Haines D:
Pathophysiology of lesion formation by transcatheter radiofrequency ablation.
In: Stephen Huang (ed): Radiofrequency catheter ablation of cardiac arrhythmias. Basic
concepts and clinical applications. New York, Futura Publishing, 1995; 25-40.
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2.
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Barhati S, Lev M:
Pathological observations of radiofrequency catheter ablation of cardiac tissue.
In: Stephen Huang (ed): Radiofrequency catheter ablation of cardiac arrhythmias. Basic
concepts and clinical applications. New York, Futura Publishing, 1995; 41-81.
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3.
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Stajzel J, Viñolas X, Boveda S, Oter R, Torner P,
Sobral J, Dumaresq L, Bayes de Luna:
Heart rate variability after radiofrequency ablation.
ANE 1997.
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4.
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Jonshon LL, Seldin DW, Becker LC, et al:
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5.
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6.
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Volpini M, Giubibini R, Gei P, et al:
Diagnosis of acute myocardial infarction by Indium-111-antimyosin antibodies and
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7.
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Carrio I, Berná L, Ballester M, et al:
Indium-111-antimyosin scintigraphy to assess myocardial damage in patients with suspected
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8.
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Obrador D, Ballester M, Carrio, et al:
High prevalence of myocardial monoclonal antibody uptake in patients with chronic
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9.
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Ballester M, Obrador D, Carrió I, et al:
Indium-111-monoclonal antimyosin antibody studies after the first year of heart
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10.
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Cossio FG, Lopez-Gil M, Goicolea A, et al:
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flutter.
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11.
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Marti V, Ballester M, Udina C, et al:
Evaluation of myocardial cel damage by In-111-monoclonal antimyosin antibodies in patients
under chronic tricyclic antidepressant drug treatment.
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