13th International Congress
THE "NEW FRONTIERS"
OF ARRHYTHMIAS 1998

January 24-31, 1998
Marilleva, Trento, Italy

RT-225

Neuronal degeneration localized in the thoracic spinal cord sympathetic center in a case with prolonged long QT interval

Lino Rossi, Luigi Matturri.
Institutes of Pathology, University of Milan, IRCCS Ospedale Maggiore, Milan, Italy

References

1.

Ashiba K, Moss AJ, Schwartz PJ (eds):
QT prolungation and ventricular arrhythmias.
Ann NY Acad Sci 1992; 644.

2.

Schwartz PJ, Priori SG, Locati EH, Napolitano C, Cantů F, Towbin JA, Keating MT, Hammoude H, Brown AM, Chen L-SK, Colatsky TJ:
Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channels blockade and to increases in heart rate: implications for gene-specific therapy.
Circulation 1995; 92: 3381-3386.

3.

Rosen MR:
Long QT syndrome patients with gene mutations.
Circulation 1995; 92: 3373-3374.

4.

Shimizu W, Antzelevitch C:
Sodium channel block with mexiletine is effective in reducing torsade de pointes in LQT2 as well as LQT3 models of long QT syndrome (abstract).
NASPE 1997; 736-738.

5.

Karjalainen J, Reunanen A, Ristola P, Vitsaalo M:
QT interval as a cardiac risk factor in a middle aged population.
Heart 1997; 77: 543-548.

6.

Karjalainen J, Vitsaalo M, Manttari M, Manninen V:
Relation between QT intervals and heart rates from 40 to 120 beats/min in rest electrocardiograms of men and a simple method to adjust QT interval values.
J Am Coll Cardiol 1994; 23: 1543-1547.

7.

Diederich KW, Djonlagic H, Schreiner WD:
Hereditares QT-syndrom: ein weiterer Fall mit grenzstrang-ganglionitis.
Herz Kreislauf 1982; 3: 149-153.

 

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